Deliriant: Difference between revisions

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It is theorized that [[choline|cholinergics]] (such as [[racetams]]) can provide relief from the mechanisms of anticholinergics,<ref>The use of a scopolamine model to study the potential nootropic effects of aniracetam and piracetam in healthy volunteers. | http://www.ncbi.nlm.nih.gov/pubmed/22281851</ref> such as reversing their [[Amnesia|amnesiatic]] and [[psychosis]]-inducing effects. This is possible by restoring typical concentrations of acetylcholine.<ref>Reversal of scopolamine-induced amnesia and alterations in energy metabolism by the nootropic piracetam: implications regarding identification of brain structures involved in consolidation of memory traces. | http://www.ncbi.nlm.nih.gov/pubmed/3690290</ref><ref>Effects of acute doses of oxiracetam in the scopolamine model of human amnesia. | http://www.ncbi.nlm.nih.gov/pubmed/7870912</ref>

In contrast, [[cannabis]] and [[caffeine]] have a reverse effect by inhibiting acetylcholine esterate, an enzyme responsible for the breakdown of acetylcholine. This leads to ~~increase in~~ levels of acetylcholine, and, in turn, improved cognition and stimulation; as a result, both [[cannabis]] and [[caffeine]] have an inhibitory effect on deliriants.

In contrast, [[cannabis]] and [[caffeine]] have a reverse effect by inhibiting acetylcholine esterate, an enzyme responsible for the breakdown of acetylcholine. This leads to increased levels of acetylcholine, and, in turn, improved cognition and stimulation; as a result, both [[cannabis]] and [[caffeine]] have an inhibitory effect on deliriants.

==Subjective effects==

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 It is theorized that [[choline|cholinergics]] (such as [[racetams]]) can provide relief from the mechanisms of anticholinergics,<ref>The use of a scopolamine model to study the potential nootropic effects of aniracetam and piracetam in healthy volunteers. | http://www.ncbi.nlm.nih.gov/pubmed/22281851</ref> such as reversing their [[Amnesia|amnesiatic]] and [[psychosis]]-inducing effects. This is possible by restoring typical concentrations of acetylcholine.<ref>Reversal of scopolamine-induced amnesia and alterations in energy metabolism by the nootropic piracetam: implications regarding identification of brain structures involved in consolidation of memory traces. | http://www.ncbi.nlm.nih.gov/pubmed/3690290</ref><ref>Effects of acute doses of oxiracetam in the scopolamine model of human amnesia. | http://www.ncbi.nlm.nih.gov/pubmed/7870912</ref>
-In contrast, [[cannabis]] and [[caffeine]] have a reverse effect by inhibiting acetylcholine esterate, an enzyme responsible for the breakdown of acetylcholine. This leads to increase in levels of acetylcholine, and, in turn, improved cognition and stimulation; as a result, both [[cannabis]] and [[caffeine]] have an inhibitory effect on deliriants.
+In contrast, [[cannabis]] and [[caffeine]] have a reverse effect by inhibiting acetylcholine esterate, an enzyme responsible for the breakdown of acetylcholine. This leads to increased levels of acetylcholine, and, in turn, improved cognition and stimulation; as a result, both [[cannabis]] and [[caffeine]] have an inhibitory effect on deliriants.
 ==Subjective effects==