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Acetylcholine: Difference between revisions
>Catharsis m Added Choline in see also. |
>Catharsis Added precursor section. |
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Other substances like [[racetam]]s can preserve acetylcholine regulation in instances of cognitive impairment such as [[deliriant]] intoxication or cognitive deficits associated with traumatic brain injury. | Other substances like [[racetam]]s can preserve acetylcholine regulation in instances of cognitive impairment such as [[deliriant]] intoxication or cognitive deficits associated with traumatic brain injury. | ||
====[[Precursors]]==== | |||
Precursors such as [[choline]], [[citicoline]] and [[alpha-GPC]] are direct building blocks for acetylcholine and supplementing them may allow it to accumulate at higher levels than that which it otherwise would.<ref>Zeisel, S. H., & Da Costa, K. A. (2009). Choline: An essential nutrient for public health. Nutrition Reviews, 67(11), 615–623. https://doi.org/10.1111/j.1753-4887.2009.00246.x</ref> | |||
====[[Antagonists]]==== | ====[[Antagonists]]==== | ||
An acetylcholine receptor [[antagonist]] works by attaching to acetylcholine receptors to prevent agonists binding. The [[deliriant]] drugs [[atropine]], [[scopolamine]] and [[diphenhydramine]] all act as antagonists upon muscarinic receptors, as does the chemical incapacitating agent 3-Quinuclidinyl benzilate, better known as BZ. | An acetylcholine receptor [[antagonist]] works by attaching to acetylcholine receptors to prevent agonists binding. The [[deliriant]] drugs [[atropine]], [[scopolamine]] and [[diphenhydramine]] all act as antagonists upon muscarinic receptors, as does the chemical incapacitating agent 3-Quinuclidinyl benzilate, better known as BZ. | ||