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Talk:T Butanol: Difference between revisions
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All claims in the section are not from sources and simply educated assumptions. It is assumed t butanol binds to the GABA A receptor producing its psychoactive effects. Unlike primary and secondary alcohols, its toxicity is lower due to it being a tertiary alcohol and not oxidised by alcohol dehydrogenase to toxic carbonyls. | All claims in the section are not from sources and simply educated assumptions. It is assumed t butanol binds to the GABA A receptor producing its psychoactive effects. Unlike primary and secondary alcohols, its toxicity is lower due to it being a tertiary alcohol and not oxidised by alcohol dehydrogenase to toxic carbonyls. | ||
==Subjective effects== | ==Subjective effects== | ||
Subjective effects of t butanol are close to ethanol but more sedating. | |||
{{Preamble/SubjectiveEffects}} | {{Preamble/SubjectiveEffects}} | ||