Talk:Internal hallucination: Difference between revisions

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:I agree with that. Thank you for adding the task to [[Talk:Subjective_effect_index]]. --[[User:David Hedlund|David Hedlund]] ([[User talk:David Hedlund|talk]]) 04:53, 19 December 2017 (CET)

==Neurological Analysis==

===Internal hallucinations are induced by a lack of external stimuli due to hallucinogens impairing your ability to discriminate external stimuli===

“The dysbalance may reflect a shift away from external stimulus-driven toward internal-driven information processing and therefore may be a determining factor for the formation of visual hallucinations. In line with this view, several computational models postulate that an increase in the excitability of the visual network in the absence of visual input destabilizes spontaneous neuronal activity, which results in the formation of elementary visual hallucinations”<ref>Kometer, M., Schmidt, A., Jäncke, L., & Vollenweider, F. X. (2013). Activation of serotonin 2A receptors underlies the psilocybin-induced effects on α oscillations, N170 visual-evoked potentials, and visual hallucinations. Journal of Neuroscience, 33(25), 10544-10551. https://doi.org/10.1523/JNEUROSCI.3007-12.2013</ref>

p. 255 “Consequently, augmentation of sensory information occurs and there are multisensory aberrations marked by fluctuations in evoked potentials, resulting in events characterized as increased distractibility. Behaviorally, this translates into an impairment in discrimination based on external stimuli and a preoccupation with internal imagery and hallucinations.“<ref>Siegel, R. K. (1985). LSD hallucinations: from ergot to electric kool-aid. Journal of Psychoactive Drugs, 17(4), 247-256. https://doi.org/10.1080/02791072.1985.10524329</ref>

p. 2558 “The nonprimary visual areas strongly modulated by Ayahuasca (BA7, BA18, and BA19) are known to be activated during psychopathological hallucinations [Allen et al., 2008] as well as during normal dreaming, within rapid-eye-movement (REM) sleep [Braun et al., 1998; Wehrle et al., 2007].“<ref>de Araujo, D. B., Ribeiro, S., Cecchi, G. A., Carvalho, F. M., Sanchez, T. A., Pinto, J. P., ... & Santos, A. C. (2012). Seeing with the eyes shut: neural basis of enhanced imagery following ayahuasca ingestion. Human brain mapping, 33(11), 2550-2560. https://doi.org/10.1002/hbm.21381</ref>

===References===

@@ Line 16: / Line 16: @@
 :I agree with that. Thank you for adding the task to [[Talk:Subjective_effect_index]]. --[[User:David Hedlund|David Hedlund]] ([[User talk:David Hedlund|talk]]) 04:53, 19 December 2017 (CET)
+==Neurological Analysis==
+===Internal hallucinations are induced by a lack of external stimuli due to hallucinogens impairing your ability to discriminate external stimuli===
+“The dysbalance may reflect a shift away from external stimulus-driven toward internal-driven information processing and therefore may be a determining factor for the formation of visual hallucinations. In line with this view, several computational models postulate that an increase in the excitability of the visual network in the absence of visual input destabilizes spontaneous neuronal activity, which results in the formation of elementary visual hallucinations”<ref>Kometer, M., Schmidt, A., Jäncke, L., & Vollenweider, F. X. (2013). Activation of serotonin 2A receptors underlies the psilocybin-induced effects on α oscillations, N170 visual-evoked potentials, and visual hallucinations. Journal of Neuroscience, 33(25), 10544-10551. https://doi.org/10.1523/JNEUROSCI.3007-12.2013</ref>
+p. 255 “Consequently, augmentation of sensory information occurs and there are multisensory aberrations marked by fluctuations in evoked potentials, resulting in events characterized as increased distractibility. Behaviorally, this translates into an impairment in discrimination based on external stimuli and a preoccupation with internal imagery and hallucinations.“<ref>Siegel, R. K. (1985). LSD hallucinations: from ergot to electric kool-aid. Journal of Psychoactive Drugs, 17(4), 247-256. https://doi.org/10.1080/02791072.1985.10524329</ref>
+p. 2558 “The nonprimary visual areas strongly modulated by Ayahuasca (BA7, BA18, and BA19) are known to be activated during psychopathological hallucinations [Allen et al., 2008] as well as during normal dreaming, within rapid-eye-movement (REM) sleep [Braun et al., 1998; Wehrle et al., 2007].“<ref>de Araujo, D. B., Ribeiro, S., Cecchi, G. A., Carvalho, F. M., Sanchez, T. A., Pinto, J. P., ... & Santos, A. C. (2012). Seeing with the eyes shut: neural basis of enhanced imagery following ayahuasca ingestion. Human brain mapping, 33(11), 2550-2560. https://doi.org/10.1002/hbm.21381</ref>
+===References===
+<references/>